Dr. Abdel Ali Belaidi completed a diploma in Biology and a PhD in Biochemistry at the University of Cologne, Germany. Dr. Belaidi studied molybdenum and sulphur amino acid metabolism and associated neurodegenerative diseases.
During his PhD and postdoctoral research in Germany, he contributed to the development of a successful treatment of the metabolic disorder molybdenum cofactor deficiency and identified new targets for the treatment of sulphite-dependent neuronal cell death.
Dr. Belaidi joined the group of Professor Ashley Bush at the Florey institute in 2014 after receiving an international Postdoctoral Fellowship from the German Research Foundation (DFG) for investigating the link between iron and neurodegenerative diseases. Dr. Belaidi is currently holding an Alzheimer’s Association research grant for investigation of Apolipoprotein E and iron association with Alzheimer’s disease.
Awards and achievements
2016- International Alzheimer's Association Research Fellowship (Chicago, United States)
2014- International Postdoctoral fellowship of the German Research Foundation (Bonn, Germany)
Research Papers
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The Complex Role of Apolipoprotein E in Alzheimer's Disease: an Overview and Update.
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Molybdenum Cofactor in HumansMolecular, Genetic, and Nutritional Aspects of Major and Trace Minerals
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Low Potential Catalytic Voltammetry of Human Sulfite OxidaseElectrochimica Acta
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Mediated Catalytic Voltammetry of Holo and Heme-Free Human Sulfite OxidasesChemElectroChem
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Chitosan-Promoted Direct Electrochemistry of Human Sulfite OxidaseThe Journal of Physical Chemistry B
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S-sulfocysteine/NMDA receptor-dependentsignaling underlies neurodegeneration in molybdenum cofactor deficiency.The Journal of clinical investigation
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Marked Age-Related Changes in Brain Iron Homeostasis in Amyloid Protein Precursor Knockout Mice.Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics
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Cellular Senescence and Iron Dyshomeostasis in Alzheimer’s DiseasePharmaceuticals
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CuII (atsm) inhibits ferroptosis: Implications for treatment of neurodegenerative disease.British journal of pharmacology
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Acute phase markers in CSF reveal inflammatory changes in Alzheimer's disease that intersect with pathology, APOE ε4, sex and age.Progress in neurobiology
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